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In this study, although we proved that ex vivo nitric oxide- and endothelium-related vasodilation in normal large vessel was induced only by BPC 157 at high concentration, it could not be ruled out that BPC 157 at low concentration might counteract the imbalanced blood pressure under an in vivo diseased condition through a currently unknown mechanism

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Bypasses first-pass hepatic metabolism entirely, producing faster CNS penetration but similar systemic clearance timelines due to mucociliary clearance and enzymatic degradation in nasal mucosa