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Dietary AX supplementation significantly alters hepatic and muscular fatty acid profiles, reducing hepatic arachidonic acid (ARA) and long-chain n -3 PUFA such as eicosapentaenoic acid (EPA), while increasing precursor fatty acids including linoleic (18:2 n -6) and α-linolenic acids (18:3 n -3), without significantly affecting Docosahexaenoic acid (DHA) levels [81].These changes are accompanied by a marked separation of hepatic fatty acid profiles in response to higher AX intake, indicating a dose-dependent remodeling of lipid metabolism